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Fête de l'écotourisme - Safari Nature - Jugon-les-lacs

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Dans le cadre de la Fête de l’écotourisme avec les labels Station Verte et Station Pêche, la commune de Jugon-les-lacs Commune Nouvelle propose des animations en mai et juin.
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Radiografía de la pelvis: MedlinePlus enciclopedia médica

Radiografía de la pelvis: MedlinePlus enciclopedia médica
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Radiografía de la pelvis
Dirección de esta página: //medlineplus.gov/spanish/ency/article/003809.htm
Radiografía de la pelvis
Para usar las funciones de compartir de esta páginas, por favor, habilite JavaScript.
Una radiografía de la pelvis es una imagen de los huesos de ambas caderas y alrededor de ellas. La pelvis conecta las piernas al cuerpo.
Forma en que se realiza el examen
El examen lo realiza un técnico en rayos X en una sala de radiología o en el consultorio del proveedor de atención médica.
Usted se acostará sobre la mesa. Luego se toman las radiografías. Es posible que tenga que mover su cuerpo a otras posiciones para proporcionar diferentes planos.
Preparación para el examen
Infórmele al proveedor si está embarazada. Quítese todas las joyas, especialmente alrededor del abdomen y las piernas. Usted usará una bata de hospital.
Lo que se siente durante el examen
Las radiografías son indoloras. Cambiar de posición puede causar molestias.
Razones por las que se realiza el examen
La radiografía se utiliza para detectar:
Fracturas
Tumores
Afecciones degenerativas de los huesos en la cadera, la pelvis y la parte superior de las piernas
Forma anormal de los huesos o articulaciones
Significado de los resultados anormales
Los resultados anormales pueden sugerir:
Fracturas pélvicas
Artritis de la articulación de la cadera
Tumores de los huesos de la pelvis
Sacroilitis (inflamación de la zona donde el sacro se une con el hueso ilion)
Espondilitis anquilosante (rigidez anormal de la columna y la articulación)
Artritis en la columna baja
Anormalidad en la forma de la pelvis o la articulación de la cadera
Riesgos
Los niños y los fetos de mujeres embarazadas son más sensibles a la radiación de las radiografías. Se puede utilizar un escudo protector sobre las zonas que no se están examinando o se pueden utilizar otras modalidades de diagnóstico por imagen.
Nombres alternativos
Radiografía - pelvis
Imágenes
Sacro
Anatomía esquelética anterior
Referencias
Beebe MJ. Fractures of the acetabulum and pelvis. In: Azar FM, Beaty JH, eds. Campbell's Operative Orthopaedics. 14th ed. Philadelphia, PA: Elsevier; 2021:chap 56.
Kapoor G, Toms AP. Current status of imaging of the musculoskeletal system. In: Adam A, Dixon AK, Gillard JH, Schaefer-Prokop CM, eds. Grainger & Allison's Diagnostic Radiology: A Textbook of Medical Imaging. 7th ed. Philadelphia, PA: Elsevier; 2021:chap 38.
Williams KD. Spondylolisthesis. In: Azar FM, Beaty JH, eds. Campbell's Operative Orthopaedics. 14th ed. Philadelphia, PA: Elsevier; 2021:chap 40.
Ultima revisión 4/1/2025
Versión en inglés revisada por: Jason Levy, MD, FSIR, Northside Radiology Associates, Atlanta, GA. Also reviewed by David C. Dugdale, MD, Medical Director, Brenda Conaway, Editorial Director, and the A.D.A.M. Editorial team.
Traducción y localización realizada por: DrTango, Inc.
Conozca cómo citar esta página
Temas de salud relacionados
Chequeo médico de la mujer
Dolor pélvico
Espondilitis anquilosante
Lesiones y enfermedades de la cadera
Lesiones y enfermedades de la pierna
Rayos X
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Snacks and sweetened drinks - children: MedlinePlus Medical Encyclopedia

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Snacks and sweetened drinks - children
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Choosing healthy snacks and drinks for your children can be hard. There are many options. What is healthy for your child may depend on specific health conditions they have.
Snacks
Fruits and vegetables are good choices for healthy snacks. They are full of vitamins and do not have added sugar or sodium. Some types of crackers and cheeses also make good snacks. Other healthy snack choices include:
Apples (fresh and cut into wedges or dried without added sugars)
Bananas
Trail mix with raisins and unsalted nuts
Chopped fruit dipped in yogurt
Raw vegetables with hummus
Carrots (regular carrots cut into strips so they are easy to chew, or baby carrots)
Snap peas (the pods are edible)
Nuts (if your child is not allergic)
Dry cereal (if sugar is not listed as one of the first 2 ingredients)
Pretzels
String cheese
Put snacks in small containers so they are easy to carry in a pocket or backpack. Use small containers to help avoid overly large portions.
Avoid having "junk food" snacks like chips, candy, cake, cookies, and ice cream every day. It is easier to keep kids away from these foods if you do not have them in your house and they are a special treat instead of an everyday item.
It is OK to let your child have an unhealthy snack once in a while. Children may try to sneak unhealthy food if they are never allowed to have these foods. The key is balance.
Other things you can do include:
Replace your candy dish with a fruit bowl.
If you have foods like cookies, chips, or ice cream in your house, store them where they are hard to see or reach. Move healthier foods to the front of the pantry and refrigerator, at eye level.
If your family snacks while watching TV, put a portion of the food in a bowl or on a plate for each person. It is easy to overeat straight from the package.
If you are not sure if a snack is healthy, read the Nutrition Facts label.
Look closely at the portion size on the label. It is easy to eat more than this amount.
Avoid snacks that list sugar as one of the first ingredients.
Try to choose snacks without added sugar or added sodium.
Beverages
Encourage children to drink a lot of water.
Avoid sodas, sport drinks, and flavored waters.
Limit drinks with added sugar. These may be high in calories and can contribute to undesired weight gain.
Prepare fruit infused water by steeping strawberries or orange slices in a pitcher for a few hours. You will get a flavored drink with virtually no sugar.
If needed, choose beverages with artificial (man-made) sweeteners.
Even 100% juices can lead to undesired weight gain. A child drinking a 12-ounce (360 milliliters) orange juice every day, in addition to other foods, can gain up to 15 excess pounds (7 kilograms) per year in addition to weight gain from normal growth patterns. Try diluting juices and flavored drinks with water. Start by adding only a little water. Then slowly increase the amount.
Children ages 1 to 6 should drink no more than 4 to 6 ounces (120 to 180 milliliters) of 100% fruit juice a day.
Children ages 7 to 18 should drink no more than 8 to 12 ounces (240 to 360 milliliters) of 100% fruit juice a day.
Children, ages 2 to 8, should drink about 2 cups (480 milliliters) of milk a day. Children older than 8 should have about 3 cups (720 milliliters) a day. It may be helpful to serve milk with meals and water between meals and with snacks.
Other Tips to Keep in Mind
The size of a snack should be the right size for your child. For example, give one half a banana to a 2-year-old and a whole banana to a 10-year-old.
Pick foods that are high in fiber and low in added salt and sugar.
Offer children fruits, vegetables, and whole-grain snacks instead of sweets.
Foods that are naturally sweet (such as apple slices, bananas, bell peppers, or baby carrots) are better than foods and drinks that contain added sugar.
Limit fried foods like French fries, onion rings, and other fried snacks.
Talk to a nutritionist or your family's health care provider if you need ideas for healthy foods for your family.
Images
Fruits and vegetables
References
Barrett KJ, Gilley SP, Haemer MA, Krebs NF, Johnson SL. Feeding healthy infants, children, and adolescents. In: Kliegman RM, St. Geme JW, Blum NJ, et al, eds. Nelson Textbook of Pediatrics. 22nd ed. Philadelphia, PA: Elsevier; 2025:chap 61.
Gahagan S. Overweight and obesity. In: Kliegman RM, St. Geme JW, Blum NJ, et al, eds. Nelson Textbook of Pediatrics. 22nd ed. Philadelphia, PA: Elsevier; 2025:chap 65.
Review Date 4/1/2025
Updated by: Stefania Manetti, RDN, CDCES, RYT200, My Vita Sana LLC - Nourish and heal through food, San Jose, CA. Review provided by VeriMed Healthcare Network. Also reviewed by David C. Dugdale, MD, Medical Director, Brenda Conaway, Editorial Director, and the A.D.A.M. Editorial team.
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内镜超声 - 医生与科室 - 妙佑医疗国际

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请参阅美国国家医学图书馆检索服务系统 PubMed 所收录的妙佑医疗国际医生在内镜超声领域的文献目录。
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Understanding EUS (endoscopic ultrasonography). Amerian Society for Gastrointestinal Endoscopy. https://www.asge.org<PATH_1> Accessed Feb. 11, 2024.
Gress FG, et al. Endoscopic ultrasound: Examination of the upper gastrointestinal tract. https://www.uptodate.com/contents/search. Accessed Feb. 11, 2024.
Wiersema MJ. Endoscopic ultrasound-guided fine needle aspiration in the gastrointestinal tract. https://www.uptodate.com/contents/search. Accessed Feb. 11, 2024.
Fasanella KE, et al. Therapeutic endoscopic ultrasound. https://www.uptodate.com/contents/search. Accessed Feb. 11, 2024.
Forbes N, et al. Adverse events associated with EUS and EUS-guided procedures. Gastrointestinal Endoscopy. 2022; doi:10.1016/j.gie.2021.09.009.
Medical review (expert opinion). Mayo Clinic. April 20, 2022.
Ami TR. Allscripts EPSi. Mayo Clinic. April 17, 2024.
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الأطباء والأقسام - Mayo Clinic (مايو كلينك)

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Marine Lantern, Solar Marine dan Lampu-lampu Navigasi Kelautan.: NEW I :Kap Lampu TIDELAND MARINE NAVIGATIONAL BUOY LANTERN BEACON ML-155, Lampu Na...

Marine Lantern, Solar Marine dan Lampu-lampu Navigasi Kelautan.: NEW I :Kap Lampu TIDELAND MARINE NAVIGATIONAL BUOY LANTERN BEACON ML-155, Lampu Navigasi Maxlumina marine Lantern
Marine Lantern, Solar Marine dan Lampu-lampu Navigasi Kelautan.
Sunergy Indonesia adalah sebuah usaha yang mengkhususkan diri bergerak dalam penjualan Marine Lantern, Solar Marine dan Lampu-lampu Navigasi Kelautan. Untuk Pemesanan/order Silahkan Tlp/sms/WA ke: 0896-2049-5107 atau email ke: <EMAIL_12> atau <EMAIL_13>
Selasa, 07 Oktober 2025
NEW I :Kap Lampu TIDELAND MARINE NAVIGATIONAL BUOY LANTERN BEACON ML-155, Lampu Navigasi Maxlumina marine Lantern
Alamat Kami :SUNERGY INDONESIA , Alamat : Kedungwinangun Rt 02 Rw 07 Pedana, Kec. Klirong, Kab. Kebumen. Jawa Tengah 54381 Tlp: 0896-2049-5107 Indonesia
Untuk Pemesanan/order Silahkan Tlp/sms/WA ke: 0896-2049-5107 atau email ke: <EMAIL_12> atau <EMAIL_13>
approx dims:
ML-155 has a 155 millimeter lens, about 6 inches in diameter.
TOTAL height is just over 19"
of which 7.5" is the gray base with the colored lens being nearly a foot tall.
Greatest total "width" is at the center where the halves meet: including the hinge protrusions, about 10".
These Lanterns are New Stock
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Kada sam videla s kim se moj muž tajno dopisuje celo telo mi se treslo: A onda mi je on otkrio istinu koju ni u najluđim snovima nisam mogla da zam...

Kada sam videla s kim se moj muž tajno dopisuje celo telo mi se treslo: A onda mi je on otkrio istinu koju ni u najluđim snovima nisam mogla da zamislim | Brze i provjerene vijesti
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Kada sam videla s kim se moj muž tajno dopisuje celo telo mi se treslo: A onda mi je on otkrio istinu koju ni u najluđim snovima nisam mogla da zamislim
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U današnnjem članku želim da podelim priču koja počinje kao bajka, ali se ubrzo pretvorila u niz neočekivanih događaja koji su moj život potpuno promenili. Moj muž i ja smo zajedno već tri godine, a u braku šest meseci.
Smatrala sam ga savršnim partnerom, čovekom kakvog sam oduvek želela, i verovala sam da živim svoj san. Međutim, ono što je trebalo da bude mirno i srećno razdoblje, odjednom je počelo da se ruši pred mojim očima.
Sve je počelo tokom jednog putovanja kolima. Nakon dva sata vožnje, stali smo da sipamo benzin i kupimo neke grickalice. Moj muž je izašao iz automobila, a ja sam ostala sama. Tada sam primetila da je njegov telefon ostao u autu i da je stigla notifikacija sa Instagrama, što me je zbunilo jer mi je ranije rekao da nema nalog i da ga ne želi. Radoznalost me je nadvladala, pa sam uzela telefon i pročitala poruku koju je primio.
Tada je počela moja unutrašnja agonija. Poruke su sadržale rečenice poput: “Volim te” i “Jedva čekam da te vidim”, a on je komentarisao slike devojke rečima poput “prelepa si”. I dok bi svaka supruga u tom trenutku osetila bol zbog sumnje u neveru, ono što me je najviše šokiralo bio je uzrast osobe kojoj su poruke upućene – samo 15, 16 godina.
Srce mi je zaledilo. Bacila sam telefon iz ruku i kada se on vratio u auto, pravila sam se da spavam, zatvarajući oči dok mi se telo treslo. Morala sam da provedem još šest sati u kolima sa čovekom kojeg sam odjednom počela da ne poznajem. Jedina uteha bila je činjenica da smo išli kod mojih roditelja, gde bih se osećala sigurnom.
Nakon dva sata putovanja, zaustavili smo se na parkingu motela. Jeza mi je prošla celim telom kada je rekao da je bolje da odmorim u pravom krevetu pre nego što nastavimo put. Po prvi put nisam se osećala sigurno pored svog muža. Povukla sam se u kupatilo, gde sam provela oko sat vremena, dok me je on molio da mu kažem šta nije u redu jer ga moje ponašanje brine.
Nisam više mogla da izdržim i rekla sam mu sve što sam saznala. On je odmah počeo da se izvinjava, objašnjavajući da je želeo da mi kaže istinu u pravom trenutku. Ali ja nisam mogla da prihvatim izvinjenje – moje misli su bile preplavljene sumnjom i šokom. Nakon nekoliko minuta tišine, rekao je da sam sve pogrešno shvatila.
Objasnio je da je devojka koja mi je izazvala šok njegova ćerka. Pokazao mi je DNK test kao dokaz njihove srodnosti. Rekao je da ga je ona prvi put kontaktirala pre godinu dana preko Facebooka. Nakon nekoliko susreta i DNK testa koji je potvrdio istinu, stvorili su jaku vezu, ali mi tada nismo znali ništa jer je bio period priprema za našu svadbu. Njegova ćerka i on nisu želeli da šire informacije dok ne budu spremni.
Priča se komplikuje jer je moj muž kao tinejdžer bio u vezi sa njenom majkom, koja je iznenada prekinula kontakt. Ćerka nije znala gde je njen otac, pa su komunicirali samo putem društvenih mreža dok nije bila spremna da otkrije istinu svojoj majci.
Otkriće me je potpuno zateklo. Nisam bila sigurna da li mogu da prihvatim ovu novu realnost i nisam odmah verovala da mi govori istinu. Moj muž je, shvativši moju sumnju, pokazao sve dokaze, uključujući poruke u kojima ga ona naziva “tata”. Suze i izvinjenja su sledile sa obe strane. Iako smo uspeli da razjasnimo nesporazum, ostala su pitanja o budućnosti našeg braka.
Dok razmišljam o svemu, ne mogu da se ne zapitam kako će ovaj događaj uticati na našu vezu i da li ćemo uspeti da prebrodimo sve izazove i sačuvamo naš brak. Situacija je i dalje sveža, ali jedno je sigurno – istina, koliko god neočekivana i šokantna, ponekad donosi olakšanje i priliku za novo razumevanje, čak i kada počinje u obliku sumnje i bola.
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Electron-impact electronic-state excitation of para-benzoquinone

Electron-impact electronic-state excitation of para-benzoquinone
Electron-impact electronic-state excitation of para-benzoquinone
J Chem Phys. 2018 Mar 28;148(12):124312. doi: 10.1063/1.5023494.
Authors
D B Jones 1 , R F da Costa 2 , F Kossoski 2 , M T do N Varella 3 , M H F Bettega 4 , F Ferreira da Silva 5 , P Limão-Vieira 5 , G García 6 , M A P Lima 2 , R D White 7 , M J Brunger 1
Affiliations
1 College of Science and Engineering, Flinders University, GPO Box 2100, Adelaide, SA 5001, Australia.
2 Instituto de Física "Gleb Wataghin," Universidade Estadual de Campinas, 13083-859 Campinas, São Paulo, Brazil.
3 Instituto de Física, Universidade de São Paulo, Rua do Matão 1731, 05508-090 São Paulo, São Paulo, Brazil.
4 Departamento de Física, Universidade Federal do Paraná, CP 19044, 81531-990 Curitiba, Paraná, Brazil.
5 Laboratório de Colisões Atómicas e Moleculares, CEFITEC, Departamento de Física, Faculdade de Ciências e Tecnologia, Universidade NOVA de Lisboa, 2829-516 Caparica, Portugal.
6 Instituto de Fisica Fundamental, CSIC, Serrano 113-bis, E-28006 Madrid, Spain.
7 College of Science and Engineering, James Cook University, Townsville, Australia.
PMID: 29604872
DOI: 10.1063/1.5023494
Abstract
Angle resolved electron energy loss spectra (EELS) for para-benzoquinone (C6H4O2) have been recorded for incident electron energies of 20, 30, and 40 eV. Measured differential cross sections (DCSs) for electronic band features, composed of a combination of energetically unresolved electronic states, are subsequently derived from those EELS. Where possible, the obtained DCSs are compared with those calculated using the Schwinger multichannel method with pseudopotentials. These calculations were performed using a minimum orbital basis single configuration interaction framework at the static exchange plus polarisation level. Here, quite reasonable agreement between the experimental cross sections and the theoretical cross sections for the summation of unresolved states was observed.


Phenotypic Plasticity: From Theory and Genetics to Current and Future Challenges - PMC

Phenotypic Plasticity: From Theory and Genetics to Current and Future Challenges - PMC
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. 2020 Mar 10;215(1):1–13. doi: 10.1534/genetics.120.303163
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Phenotypic Plasticity: From Theory and Genetics to Current and Future Challenges
Ralf J Sommer
Ralf J Sommer
1Max Planck Institute for Developmental Biology, Department for Integrative Evolutionary Biology, 72076 Tübingen, Germany
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1Max Planck Institute for Developmental Biology, Department for Integrative Evolutionary Biology, 72076 Tübingen, Germany
1
Address for correspondence: Max Planck Institute for Developmental Biology, Max-Planck Ring 9, 72076 Tübingen, Germany. E-mail: <EMAIL_10>
Received 2019 Nov 29; Accepted 2020 Mar 9; Issue date 2020 May.
Copyright © 2020 by the Genetics Society of America
Available freely online through the author-supported open access option.
PMC Copyright notice
PMCID: PMC7198268 PMID: 32371438
Phenotypic plasticity is defined as the property of organisms to produce distinct phenotypes in response to environmental variation. While for more than a century, biologists have proposed this organismal feature...
Keywords: phenotypic plasticity, polyphenisms, switch genes, plasticity first evolution, canalization, genetic assimilation, genetic accommodation, Pristionchus, Spea, Ontophagus, Manduca
Abstract
Phenotypic plasticity is defined as the property of organisms to produce distinct phenotypes in response to environmental variation. While for more than a century, biologists have proposed this organismal feature to play an important role in evolution and the origin of novelty, the idea has remained contentious. Plasticity is found in all domains of life, but only recently has there been an increase in empirical studies. This contribution is intended as a fresh view and will discuss current and future challenges of plasticity research, and the need to identify associated molecular mechanisms. After a brief summary of conceptual, theoretical, and historical aspects, some of which were responsible for confusion and contention, I will formulate three major research directions and predictions for the role of plasticity as a facilitator of novelty. These predictions result in a four-step model that, when properly filled with molecular mechanisms, will reveal plasticity as a major factor of evolution. Such mechanistic insight must be complemented with comparative investigations to show that plasticity has indeed created novelty and innovation. Together, such studies will help develop a true developmental evolutionary biology.
PHENOTYPIC plasticity is the ability of a genotype to produce different phenotypes in response to distinct environmental conditions (Schlichting and Pigliucci 1998; Pigliucci 2001; West-Eberhard 2003; deWitt and Scheiner 2004; Whitman and Ananthakrishnan 2009; Moczek et al. 2011). Intrinsically, phenotypic plasticity refers to all kinds of environmentally induced phenotypic variation and it can affect morphological, physiological, and behavioral aspects of an organism’s phenotype, but also its life history. Plasticity is a universal property of living things, because all organisms respond to genes and the environment alike; thus, plasticity is found throughout all domains of life. While botanists have long appreciated the environmental influence on plant morphology, plasticity was less valued in animal systems, although it is as widespread in animals as in plants (West-Eberhard 1989). In addition, plasticity is known from bacteria, and even phage λ and other bacteriophages with their lytic (virulent) vs. lysogenic (temperate) life cycles. It is an interesting oddity of the history of biology that the first molecular process that was ever elucidated to near completion, the regulation of the lytic cycle in phage λ, represents an example of plasticity, even though it is rarely discussed as such (Ptashne 2004).
Plasticity is pervasive, as demonstrated by the many examples currently studied in laboratories around the world. In the interest of space, I will not provide an overview of these study systems as there are simply too many. Instead, I refer the reader to the many review articles that have been published in recent years and that provide excellent overviews (Abouheif et al. 2014; Lande 2014; Laland et al. 2014, 2015; Moczek et al. 2015; Nalepa 2015; Nijhout 2015; Brisson and Davis 2016; Phillips 2016; Susoy and Sommer 2016; Tandonnet and Pires-daSilva 2016; Gibert 2017; Noble et al. 2017; Projecto-Garcia et al. 2017; Reuter et al. 2017; Schneider and Meyer 2017; Serobyan and Sommer 2017; Gilbert 2018; Jones and Robinson 2018; Josephs 2018; Oettler et al. 2018; Sanger and Rajakumar 2018; Sieriebriennikov and Sommer 2018; Uller et al. 2018; Lafuente and Beldade 2019; Levis and Pfennig 2019). While this list contains only those reviews published since 2014 and is likely still incomplete, it demonstrates the growing awareness about plasticity and its evolutionary significance.
This growing awareness is in strong contrast to a long phase of neo-Darwinism that neglected the importance of development and the significance of the organism’s responsiveness to the environment for evolution. For example, Williams objected that plasticity guarantees a dead end for the underlying traits, arguing that plasticity hinders evolution (Williams 1966). Similar arguments were made by Charlesworth et al. (1982), disputing the importance of development and the environment in favor of selection as the “main guiding force of phenotypic evolution” (Charlesworth et al. 1982, p. 474). Such skepticism remained for decades and largely centered around three major reservations (Box 1A) (Wund 2012). First, is there sufficient empirical evidence for plasticity in general, and for plasticity as a driver of evolutionary change? Second, does plasticity act to promote or hinder evolution? Finally, what could be the molecular mechanisms of the environmental influence on phenotypes, how do such mechanisms become genetically encoded, and how do they become a target of selection? These reservations highlighted the fundamental challenges for research on phenotypic plasticity, but they also provided a road map for novel investigations.
Box 1.
A: Historical skepticism against phenotypic plasticity and its significance for evolution
1. Empirical evidence for plasticity?
2. Can environmental responsiveness promote evolution?
3. Molecular mechanisms of environmental influence?
How should environmental effects be targeted by selection?
B: Developmental plasticity and evolution - West-Eberhard and four unique contributions for plasticity as a mechanism of evolution
A giant collection of alternative phenotypes
Alternative phenotypes as functionally independent targets of selection
A general critique of Neo-Darwinism and its inconsistencies and gaps
Plasticity as a facilitator of novelty (The facilitator hypothesis)
C:


NIH VideoCast - 2025 Joram Piatigorsky Basic Science Lecture and Award

NIH VideoCast - 2025 Joram Piatigorsky Basic Science Lecture and Award
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2025 Joram Piatigorsky Basic Science Lecture and Award
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Air date: Wednesday, April 23, 2025, 3:00:00 PM
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Views: Total views: 167 (63 Live, 104 On-demand)
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Description: Claude Desplan, Ph.D. from New York University, will deliver this year's 2025 Joram Piatigorsky Basic Science Lecture and Award. The lecture will take place on Wednesday, April 23, 2025, from 3:00 PM-4:15 PM at Masur Auditorium, located in the Clinical Center (Building 10) on the NIH campus. It will be followed by a light reception. Trainees and scientists from all NIH ICs are encouraged to attend. Made possible by the generous philanthropic support of Lona and Joram Piatigorsky, this series brings attention to notable basic sciences contributions by eye and vision scientists to a diverse general scientific audience, such as experts in molecular biology, genetics, developmental biology, neuroscience, and computer science. With special consideration for basic eye and vision scientists who take risks exploring little-studied species and imaginative ideas, the Lecture and Award promotes and communicates basic discoveries in eye and vision research that result in far-reaching observations that may inform widespread areas of science—from the eye to the world as it were—rather than the other way around.
Author: Claude Desplan, PhD
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Endogenous 17β-estradiol is required for activity-dependent long-term potentiation in the striatum: interaction with the dopaminergic system

Endogenous 17β-estradiol is required for activity-dependent long-term potentiation in the striatum: interaction with the dopaminergic system
Endogenous 17β-estradiol is required for activity-dependent long-term potentiation in the striatum: interaction with the dopaminergic system
Front Cell Neurosci. 2015 May 27:9:192. doi: 10.3389/fncel.2015.00192. eCollection 2015.
Authors
Alessandro Tozzi 1 , Antonio de Iure 2 , Michela Tantucci 2 , Valentina Durante 2 , Ana Quiroga-Varela 2 , Carmela Giampà 3 , Michela Di Mauro 4 , Petra Mazzocchetti 2 , Cinzia Costa 2 , Massimiliano Di Filippo 2 , Silvarosa Grassi 4 , Vito Enrico Pettorossi 4 , Paolo Calabresi 5
Affiliations
1 Department of Experimental Medicine, Section of Physiology and Biochemistry, University of Perugia Perugia, Italy ; Fondazione Santa Lucia, IRCCS Rome, Italy.
2 Clinica Neurologica, Dipartimento di Medicina, Università degli Studi di Perugia, Ospedale Santa Maria della Misericordia Perugia, Italy.
3 Fondazione Santa Lucia, IRCCS Rome, Italy.
4 Department of Experimental Medicine, Section of Physiology and Biochemistry, University of Perugia Perugia, Italy.
5 Fondazione Santa Lucia, IRCCS Rome, Italy ; Clinica Neurologica, Dipartimento di Medicina, Università degli Studi di Perugia, Ospedale Santa Maria della Misericordia Perugia, Italy.
PMID: 26074768
PMCID: PMC4445326
DOI: 10.3389/fncel.2015.00192
Abstract
17β-estradiol (E2), a neurosteroid synthesized by P450-aromatase (ARO), modulates various brain functions. We characterized the role of the locally synthesized E2 on striatal long-term synaptic plasticity and explored possible interactions between E2 receptors (ERs) and dopamine (DA) receptors in the dorsal striatum of adult male rats. Inhibition of E2 synthesis or antagonism of ERs prevented the induction of long-term potentiation (LTP) in both medium spiny neurons (MSNs) and cholinergic interneurons (ChIs). Activation of a D1-like DA receptor/cAMP/PKA-dependent pathway restored LTP. In MSNs exogenous E2 reversed the effect of ARO inhibition. Also antagonism of M1 muscarinic receptors prevented the D1-like receptor-mediated restoration of LTP confirming a role for ChIs in controlling the E2-mediated LTP of MSNs. A novel striatal interaction, occurring between ERs and D1-like receptors in both MSNs and ChIs, might be critical to regulate basal ganglia physiology and to compensate synaptic alterations in Parkinson's disease.
Keywords: D1 receptor; P450-aromatase; cholinergic interneurons; estrogen receptors; long-term potentiation; medium spiny neurons; striatum; synaptic plasticity.


The Four Pillars of Health Coaching: Preserving the Heart of a Movement - PMC

The Four Pillars of Health Coaching: Preserving the Heart of a Movement - PMC
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. 2013 May 1;2(3):6–8. doi: 10.7453/gahmj.2013.038
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The Four Pillars of Health Coaching: Preserving the Heart of a Movement
健康辅导的四大支柱：保留这项举动的核心
Los cuatro pilares de la formación sanitaria: conservación de la esencia de un movimiento
Karen Lawson
Karen Lawson, MD, ABIHM
1Center for Spirituality & Healing, University of Minnesota, Minneapolis, United States
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1Center for Spirituality & Healing, University of Minnesota, Minneapolis, United States
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Email: <EMAIL_8>
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Corresponding author.
Issue date 2013 May.
© 2013 GAHM LLC.
This is an open-access article distributed under the terms of the Creative Commons Attribution-Non Commercial- No Derivative 3.0 License, which permits rights to copy, distribute and transmit the work for noncommercial purposes only, provided the original work is properly cited.
PMC Copyright notice
PMCID: PMC3833536 PMID: 24416668
Abstract
In this special themed issue of Global Advances in Health and Medicine and in articles published on the journal's website (www.gahmj.com), you will read all about this new and maturing approach to health behavior change and the social and cultural conditions in modern medicine that have called this practice into being. You will learn about its inception and history,1 the philosophic constructs of its application,2,3 and its proposed mechanism of action.4 There are multiple case reports5–7 and clinical studies8–10 expanding our scientific understanding of coaching in health and wellness and descriptions about how to educate professionals to provide this new service.11,12 A review of existing literature in the field demonstrates the rapidly growing reported demonstration of its impact.13 I believe coaching is poised to have a major transformative impact on health and healthcare internationally; it also is at risk of being usurped and thereby deformed by the power of the existing paradigm of Western disease care and reductionistic scientific thought. That is the issue I wish to raise: How do we ensure the integrity of this new approach so that it can serve as a bridge from a broken system to a new horizon of holistic health and well-being?
Key Words: Health coaching, behavior change, Four Pillars, mind, body, spirit
In this special themed issue of Global Advances in Health and Medicine and in articles published on the journal's website (www.gahmj.com), you will read all about this new and maturing approach to health behavior change and the social and cultural conditions in modern medicine that have called this practice into being. You will learn about its inception and history,1 the philosophic constructs of its application,2,3 and its proposed mechanism of action.4 There are multiple case reports5–7 and clinical studies8–10 expanding our scientific understanding of coaching in health and wellness and descriptions about how to educate professionals to provide this new service.11,12 A review of existing literature in the field demonstrates the rapidly growing reported demonstration of its impact.13 I believe coaching is poised to have a major transformative impact on health and healthcare internationally; it also is at risk of being usurped and thereby deformed by the power of the existing paradigm of Western disease care and reductionistic scientific thought. That is the issue I wish to raise: How do we ensure the integrity of this new approach so that it can serve as a bridge from a broken system to a new horizon of holistic health and well-being?
I came to my own epiphany about the limitations and casualties of contemporary medicine in the mid-1980s during my training and early practice years as a family physician. It was a time when family practice was a newly recognized specialty and was working to prove itself as a respectable and professional branch of medicine. I was privileged to have the opportunity to learn from some of the last general practitioners who still did home visits, delivered babies, performed their own surgeries, provided care to entire multigenerational families over many years, and sat at the bedsides of the dying. They were, in many ways, the last of their kind in the United States. Through the introduction and rapid dominance of health maintenance organization models, the increasing corporatization of medicine, growing subspecialty development, and burgeoning fragmentation of care, the holism I sought became a rarity in the halls of hospitals and clinics. I began a search for other approaches to health and healing that acknowledged the mind, body, and spirit of individuals and families; empowered individuals in their own decision-making; and championed the innate healing capacities of humans when they have the support and resources they need. That search led me to learn about mind-body practices, functional nutrition, movement practices, energy medicine, systems such as traditional Chinese medicine and homeopathy, and spiritual healing approaches.
By the late 1990s, I had become an advocate and leader of holistic medicine and shifted my professional energies to that field, hoping to transform health-care from inside of medicine. In the process of leading the development of new interdisciplinary team models for holistic clinics, it became clear to me that there was a missing provider. Whether an individual received expert guidance from doctor of medicine, a doctor of chiropractic, a naturopathic doctor, a physical therapist, or a licensed acupuncturist, the patient often left the clinic with a great deal of information about changes to make and no clear plan as to how to make them. Patients needed someone to know and understand their unique strengths, challenges, and desires—someone to walk with them through the demanding process of implementing new beliefs and behaviors in their lives—in a way that would promote their own health and healing at the pace for which they were ready. This person needed to be in a trusted relationship with the patient, to be readily and frequently available, to see the patient as the capable expert in his or her own life, to understand the nature of the change process, and to be able to hold the vision of greater health for and with the patient. This person was, and is, an integrative health coach.
When I joined the University of Minnesota Center for Spirituality and Healing in 2002, one of the topics of exploration was this: How do we create a training program for these new providers? Who would they be? How and in which subjects did they need to be educated? For the early clinical models I had been involved with, we had identified unique and talented individuals with a passion for this work. With the support and guidance of pioneers in the field such as Linda Bark, PhD, RN, MCC, NC-BC, we trained and supervised these new providers in


Maia Fraser | OpenReview

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Examen de piel - PPD (brazo derecho) y Candida (izquierdo): MedlinePlus enciclopedia médica illustración

Examen de piel - PPD (brazo derecho) y Candida (izquierdo): MedlinePlus enciclopedia médica illustración
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Examen de piel - PPD (brazo derecho) y Candida (izquierdo)
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El brazo derecho de esta persona presenta una reacción positiva a la PPD (prueba de la piel para la proteína de la tuberculosis). El brazo izquierdo presenta una reacción positiva a la proteína de Candida. Se prueba el antígeno de Candida para determinar si el sistema inmunitario de la persona está funcionando bien; un sistema inmunitario normal muestra una reacción positiva.
Ultima revisión 3/31/2024
Versión en inglés revisada por: Deborah Pedersen, MD, MS, Allergy & Asthma Care, PC, Taunton, MA. Review provided by VeriMed Healthcare Network. Also reviewed by David C. Dugdale, MD, Medical Director, Brenda Conaway, Editorial Director, and the A.D.A.M. Editorial team.
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DailyMed - ELETRIPTAN HBR tablet, film coated

DailyMed - ELETRIPTAN HBR tablet, film coated
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Label: ELETRIPTAN HBR tablet, film coated
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NDC Code(s): 72189-439-06
Packager: Direct_Rx
This is a repackaged label.
Source NDC Code(s): 27241-040
Category: HUMAN PRESCRIPTION DRUG LABEL
Drug Label Information
Updated January 22, 2025
If you are a healthcare professional or from the pharmaceutical industry please visit this version.
Download DRUG LABEL INFO: PDF XML
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INDICATIONS & USAGE
Eletriptan hydrobromide tablets are indicated for the acute treatment of migraine with or without aura in adults.
Limitations of Use:
Use only if a clear diagnosis of migraine has been established. If a patient has no response to the first migraine attack treated with eletriptan hydrobromide tablets, reconsider the diagnosis of migraine before eletriptan hydrobromide tablets are administered to treat any subsequent attacks.
Eletriptan hydrobromide tablets are not intended for the prevention of migraine attacks.
Safety and effectiveness of eletriptan hydrobromide tablets have not been established for cluster headache.
DOSAGE & ADMINISTRATION
The maximum recommended single dose is 40 mg.
In controlled clinical trials, single doses of 20 mg and 40 mg were effective for the acute treatment of migraine in adults. A greater proportion of patients had a response following a 40 mg dose than following a 20 mg dose [see Clinical Studies (14)].
If the migraine has not resolved by 2 hours after taking eletriptan hydrobromide tablets, or returns after transient improvement, a second dose may be administered at least 2 hours after the first dose. The maximum daily dose should not exceed 80 mg.
The safety of treating an average of more than 3 migraine attacks in a 30-day period has not been established.
DOSAGE FORMS & STRENGTHS
20 mg Tablets: Orange, round, biconvex, film-coated, debossed with “E1” on one side and plain on another side.
40 mg Tablets: Orange, round, biconvex, film-coated, debossed with “E2” on one side and plain on another side.
CONTRAINDICATIONS
Eletriptan hydrobromide tablets are contraindicated in patients with:
Ischemic coronary artery disease (CAD) (angina pectoris, history of myocardial infarction, or documented silent ischemia) or coronary artery vasospasm, including Prinzmetal’s angina [see Warnings and Precautions (5.1)].
Wolff-Parkinson-White syndrome or arrhythmias associated with other cardiac accessory conduction pathway disorders [see Warnings and Precautions (5.2)].
History of stroke, transient ischemic attack (TIA), or history or current evidence of hemiplegic or basilar migraine because these patients are at a higher risk of stroke [see Warnings and Precautions (5.4)].
Peripheral vascular disease [see Warnings and Precautions (5.5)].
Ischemic bowel disease [see Warnings and Precautions (5.5)].
Uncontrolled hypertension [see Warnings and Precautions (5.8)].
Recent use (i.e., within 24 hours) of another 5-hydroxytryptamine1 (5-HT1) agonist, ergotamine-containing medication, or ergot-type medication such as dihydroergotamine (DHE) or methysergide [see Drug Interactions (7.1)].
Hypersensitivity to eletriptan hydrobromide tablets (angioedema and anaphylaxis seen) [see Warnings and Precautions (5.9)].
Recent use (i.e., within at least 72 hours) of the following potent CYP3A4 inhibitors: ketoconazole, itraconazole, nefazodone, troleandomycin, clarithromycin, ritonavir, or nelfinavir [see Drug Interactions (7.2) and Clinical Pharmacology (12.3)].
WARNINGS AND PRECAUTIONS
Eletriptan hydrobromide tablets should only be used where a clear diagnosis of migraine has been established.
5.1 Myocardial Ischemia, Myocardial Infarction, and Prinzmetal's Angina
Eletriptan hydrobromide tablets are contraindicated in patients with ischemic or vasospastic CAD. There have been rare reports of serious cardiac adverse reactions, including acute myocardial infarction, occurring within a few hours following administration of eletriptan hydrobromide tablets. Some of these reactions occurred in patients without known CAD. Eletriptan hydrobromide tablets may cause coronary artery vasospasm (Prinzmetal’s angina), even in patients without a history of CAD.
Perform a cardiovascular evaluation in triptan-naive patients who have multiple cardiovascular risk factors (e.g., increased age, diabetes, hypertension, smoking, obesity, strong family history of CAD) prior to receiving eletriptan hydrobromide tablets. Do not use eletriptan hydrobromide tablets if there is evidence of CAD or coronary artery vasospasm [see Contraindications (4)]. For patients with multiple cardiovascular risk factors who have a negative cardiovascular evaluation, consider administering the first eletriptan hydrobromide tablets dose in a medically-supervised setting and performing an electrocardiogram (ECG) immediately following administration of eletriptan hydrobromide tablets. For such patients, consider periodic cardiovascular evaluation in intermittent long-term users of eletriptan hydrobromide tablets.
5.2 Arrhythmias
Life-threatening disturbances of cardiac rhythm including ventricular tachycardia and ventricular fibrillation leading to death have been reported within a few hours following the administration of 5-HT1 agonists. Discontinue eletriptan hydrobromide tablets if these disturbances occur. Eletriptan hydrobromide tablets are contraindicated in patients with Wolff-Parkinson-White syndrome or arrhythmias associated with other cardiac accessory conduction pathway disorders [see Contraindications (4)].
5.3 Chest, Throat, Neck and/or Jaw Pain/Tightness/Pressure
Sensations of tightness, pain, and pressure in the chest, throat, neck, and jaw commonly occur after treatment with eletriptan hydrobromide tablets and are usually non-cardiac in origin. However, perform a cardiac evaluation if these patients are at high cardiac risk. Eletriptan hydrobromide tablets are contraindicated in patients with CAD or Prinzmetal’s variant angina [see Contr